Reprinted with the permission of itslyme. As an active kid growing up in New England, I was often playing outdoors behind our house, in parks, and on the soccer field. Two days later, my head was still sore. My aunt took a look, and noticed there was a tick embedded just above my hairline. The tick was extracted with a pair of tweezers, and nothing else was done.
I was devastated. Lyme disease is usually treated with medication for no longer than 28 days. When these bacteria infect the nervous system, the infection is called neuroborreliosis, Timothy J. Eighty per cent of reported cases were from nine northeast and midatlantic states. Serodiagnosis in early Lyme disease.
Female redheads. References
Kenneth B. Get Started. Debby Cheboygan, Michigan, United States. Check Your Symptoms. Who Suffers Facial Nerve Damage? Send Tired of Lyme a list of your symptoms. This may also occur when early treatment is inadequate. Neurological exam Electromyography Nerve conduction velocity test Blood testsincluding tests for antibody against the agent that causes Lyme disease and tests to Lyme disease facial twitches dizziness nausea the agent itself. Donate Now. Facial paralysis Facial muscle weakness Loss of taste and smell Dry eyes. You may also need to have some blood tests and scans to determine the actual cause of the facial nerve damage. In some cases, a doctor will prescribe medication to reduce the inflammation of the facial nerve. My Protocol. Your email address will not be published.
Not all tick removal methods are safe.
- Home Introduction.
- Home Introduction.
- Various medical conditions can cause facial nerve damage, resulting in a range of symptoms developing.
- New To Lyme?
This past summer, Lyme disease was the topic of a Focus section in the Globe and Mail 1. In this section, the reporter described her experience of having physicians unable and then unwilling to diagnose her symptoms of "skin on fire, dizziness and chest pains, twitching muscles, and trouble keeping balance" as Lyme disease following a tick bite three years previously on Prince Edward Island.
She reported finding support for her diagnosis after obtaining a positive test from a California laboratory and after seeing approximately 20 physicians. In her article, she speaks to the controversy surrounding the diagnosis and treatment of Lyme disease, and the tension it creates between those who believe they have it and the physicians they see. Controversy is not new to medicine, and the Lyme disease controversy is a case in point.
In the United States US , the lay press has given the term 'Lyme camps' to describe the two opposing views, held on the one hand by academic-based physicians who use limited-duration antibiotic therapy to treat Lyme disease, and on the other hand by community-based physicians and advocacy groups who believe in open-ended duration antimicrobials to treat Lyme disease 2.
The Infectious Diseases Society of America was compelled to write to the US House of Representatives in August , cautioning against passing legislation that might allow for the prolonged antibiotic treatment for Lyme disease, which is not supported by the best available evidence personal communication.
Lyme disease was first recognized 30 years ago by Allen Steere and colleagues as an epidemic of oligoarticular arthritis in children and adults in three Connecticut communities 3. His group of investigators, and another from New York State, concluded that a spirochete, Borrelia burgdorferi , carried by the Ixodes dammini tick was the etiological agent of Lyme disease after isolating this organism from skin, cerebrospinal fluid and blood of patients with the clinical syndrome and showing an immunological response 4 , 5.
Credit is given to two women, one a concerned mother, for bringing this epidemic of arthritis to the attention of public health officials and physicians 6. In and , there were 20, and 18, cases, respectively, reported to the Centers for Disease Control and Prevention 7.
Eighty per cent of reported cases were from nine northeast and midatlantic states. Lyme disease is also transmitted in Asia and Europe by ticks of the Ixodes ricinus complex of which Ixodes scapularis [previously classified as I dammini ] is one member , sometimes with regional variations in clinical manifestations 8. Lyme disease is not a national notifiable disease in Canada, making it very difficult to identify in any systematic way the number of cases and trends over time.
It is reportable in some provinces. Established populations of these blacklegged deer ticks are found only along the north shore of Lake Erie, particularly in Long Point, Point Pelee and Rondeau Provincial Park, in selected localities along the shores of Lake Ontario, and near Lunenburg, Nova Scotia 10 , In other areas where infected blacklegged tick populations have not been established, it is presumed that the ticks are introduced into the area by migratory birds 9 , The number of reported human cases of Lyme disease in Canada has been very small and is also available on several provincial health department Web sites.
There have been only three confirmed Nova Scotia-acquired cases to date, even with the established infected tick population near Lunenburg. The risk of developing Lyme disease after a tick bite and its clinical manifestations have been well described in the context of randomized, controlled trials of vaccination and antimicrobial prophylaxis after tick bites, as well as in longitudinal observational studies of populations from endemic areas.
Shapiro et al 12 found that 1. There were no asymptomatic seroconversions in this cohort. A slightly higher rate of infection 3. In this latter study, all transmissions were associated with bites from nymphal ticks not adult or larval and there were no transmissions in the 48 patients where the nymphal tick had been feeding for less than 72 h Again, there were no asymptomatic seroconversions in the study population.
In two vaccine studies conducted in endemic areas, attack rates for Lyme disease were 1. However, EM may be both overdiagnosed and underdiagnosed 18 , in part due to the potential for less classic manifestations None had upper respiratory or gastrointestinal tract symptoms. It is clear that a high index of suspicion for Lyme disease should be maintained when evaluating patients from endemic areas with systemic symptoms following a tick bite.
There are several different modalities for the laboratory diagnosis of Lyme disease, and these have recently been reviewed in Clinical Microbiology Reviews Culture of B burgdorferi from clinical specimens is not used in routine clinical practice for several reasons, including logistics lack of consistent, high-quality growth medium, slow growth and expense and poor sensitivity Thus, the laboratory-based diagnosis of Lyme disease is primarily an immunological one, based on detection of antibodies against various B burgdorferi antigens.
Since the etiological agent was first identified, a number of immunological tests have been developed and tested. In addition, significant inter- and intralaboratory variations in the serodiagnosis of Lyme disease have been reported 22 , A number of different commercial products are available, and band criteria for interpreting IB have been published A review of Lyme diagnostics by Aguero-Rosenfeld et al 21 summarized the findings of a number of published studies examining the serodiagnosis of Lyme.
Equally evident is that serology has poor utility in acute EM, which is a clinical diagnosis and does not require serological confirmation. Finally, antibody concentrations may remain positive for many months, even in treated patients. Despite the availability of 70 serological assays to aid in the diagnosis of Lyme disease and recommendations for the use and interpretation of these tests, the Centers for Disease Control and Prevention recently had to publish a caution regarding some commercial laboratories testing for Lyme disease with nonapproved tests or interpretive criteria In an early study 30 that compared the outcome of patients treated with penicillin with a historical cohort of nontreated patients, a day course of penicillin seemed to shorten the course of EM and reduce the risk of arthritis.
In a small randomized, controlled trial conducted in 32 , cefuroxime and doxycycline each for 20 days were equally effective in treating early Lyme disease. Lyme arthritis did not develop in any patient one month after treatment. A subsequent study 33 showed that doxycycline for 10 days is as effective as a longer course 20 days or a day course preceded by a single dose of intravenous ceftriaxone. Only one patient in that trial failed therapy and required treatment with intravenous ceftriaxone for meningitis.
There were no differences in neurocognitive testing between the three treatment groups at 30 months To examine whether prolonged antibiotic therapy would be of benefit to symptomatic patients with a documented history of treated Lyme disease, a trial comparing 30 days of intravenous ceftriaxone followed by 60 days of oral doxycycline versus placebo was begun in This trial was terminated by the data and safety monitoring board after a planned interim analysis when it became evident that it was highly unlikely there would be a difference in the two treatment groups There are no large, randomized, controlled trials comparing different regimens in the treatment of Lyme arthritis and other manifestations of Lyme disease.
On the basis of the available case series and small trials, the Infectious Diseases Society of America has published practice guidelines for the treatment of these and early manifestations of Lyme disease The persistence of symptoms in some patients treated for Lyme disease has already been noted.
This contributes, in part, to the debate surrounding the entity that has been called 'chronic Lyme'. Delayed antibiotic therapy average of These findings were corroborated in a randomized, controlled trial of treatment for post-treatment chronic Lyme 40 , in which symptomatic patients had no objective evidence of cognitive impairment.
Finally, a population-based study 41 in Connecticut found a similar frequency of Lyme symptoms such as pain and fatigue or difficulty with daily activities between patients with Lyme disease from to and age-matched controls without Lyme disease.
Steere et al 43 excluded Lyme disease in of patients referred to them for the evaluation of Lyme disease. Patients in that study without active Lyme disease had a median of four serological tests done range one to 16 before referral The potential for the recognized adverse events associated with long-term antibiotic therapy appears to be a risk patients seeking treatment for chronic Lyme are prepared to take.
Many infectious diseases physicians are not as comfortable exposing patients to these risks in the absence of evidence for the benefit of long-term antimicrobial therapy for patients suffering symptoms after completing the standard course of therapy for Lyme disease.
Concerns regarding the safety of long-term antibiotics may increase if recent research suggesting a link between prolonged antimicrobial therapy and cancer is substantiated Much has been learned about Lyme disease in only 30 short years.
Until then, physicians and patients are left to manage with the best evidence available. It is unfortunate that tension has arisen over the diagnosis and management of Lyme disease. In the end, we all want what is best for the patient. National Center for Biotechnology Information , U.
Author information Article notes Copyright and License information Disclaimer. Telephone , fax , e-mail ac. Received Nov 7; Accepted Nov 7.
All rights reserved. This article has been cited by other articles in PMC. References 1. Bourette S.
Ticking bomb? The Globe and Mail , section F1, August 20, Feder HM Jr. Differences are voiced by two Lyme camps at a Connecticut public hearing on insurance coverage of Lyme disease. Pediatrics ; Lyme arthritis: An epidemic of oligoarticular arthritis in children and adults in three connecticut communities. Arthritis Rheum ; 20 The spirochetal etiology of Lyme disease. N Engl J Med ; Spirochetes isolated from the blood of two patients with Lyme disease.
Harris ED Jr. Lyme disease - success for academia and the community. Centers for Disease Control and Prevention. Steere AC. Lyme disease: A growing threat to urban populations. Vector of Lyme borreliosis, Ixodes scapularis , identified in Saskatchewan. Can Commun Dis Rep ; 25 Public Health Agency of Canada. Lyme disease. Government of Ontario. Diseases: Lyme disease.
A controlled trial of antimicrobial prophylaxis for Lyme disease after deer-tick bites. Prophylaxis with single-dose doxycycline for the prevention of Lyme disease after an Ixodes scapularis tick bite. A vaccine consisting of recombinant Borrelia burgdorferi outer-surface protein A to prevent Lyme disease. Erratum in ; Vaccination against Lyme disease with recombinant Borrelia burgdorferi outer-surface lipoprotein A with adjuvant.
Lyme Disease Vaccine Study Group. The presenting manifestations of Lyme disease and the outcomes of treatment.
I quickly began having symptoms again but was determined and thus continued to work. Fever Skin rashes Fatigue Headaches. Can't make a fist. Submit a Comment Cancel reply Your email address will not be published. Sore muscles and joints Sore neck and hips and other parts of body on and off throughout the day. Lyme Disease Neuropathy. If you have any symptoms you should consult a doctor at the earliest opportunity.
Lyme disease facial twitches dizziness nausea. Chronic Lyme Disease Series: Kelly’s Story
Symptoms | Lyme Disease Action
Chat with us in Facebook Messenger. Find out what's happening in the world as it unfolds. It then develops into neurological problems, such as numbness in the limbs or facial paralysis, leaving the patient in excruciating pain.
About , Americans each year are infected with Lyme disease , according to the Centers for Disease Control and Prevention.
That number, the CDC reported last week , is about 10 times higher than they had previously estimated. But what if you never get the rash?
What if you blow off your symptoms as a bad case of the flu? I spent the first 30 years of my life in New England, where talk about Lyme disease was prevalent. When I was little my mother would have us slather on the DEET before we went outside made sure we had long pants and socks on, especially when we'd play in the woods. She'd check us for ticks and remove any she found with a match and tweezers. Problem solved -- no rash, good to go.
Buzz off, bugs! How to stay safe during insect season. Then in I moved to Atlanta, and Lyme disease faded from my mind. Common in the Northeast, Lyme disease is found less often in the South. The year I arrived, eight cases were reported in Georgia , compared to 1, in my home state of Connecticut. At least once a week, my muscles start to twitch uncontrollably; they tighten so much my fingers turn into fists. I can feel my fingernails digging through my skin.
My heart races out of control and my blood pressure skyrockets. My doctor holds me down, consoling me, telling me to breathe because he has no idea what's going on. My face is numb, and it feels like I have the flu. I have trouble breathing. The pain is so bad I start to cry. Avoiding ticks A few years ago I noticed a pain radiating down my leg, starting from the small of my back.
I had had artificial disc surgery in my spine a couple years prior and thought this new pain was due to scar tissue from the surgery. Then I went to work one day and lost all feeling in my legs. My arms were tingling and it felt like I had pins and needles everywhere in my body. I went to the ER, and that night, I became a medical mystery.
I had a full body MRI the first of many and by the end of the night I had seen at least five neurologists. They chalked it up to my previously damaged spine, gave me some painkillers and sent me on my way. This sequence of events would continue for another two years. Each time the diagnosis would change, but the end result would stay the same. I saw a neurologist, a neurosurgeon, an orthopedic surgeon, a urologist, a neuro-urologist, a kidney specialist, three different pain doctors and a pain psychologist.
My diagnoses varied from heart disease to polycystic kidney disease to Lupus. I had many different neurologic disorders -- fibromyalgia, psychosomatic disorder and my personal favorite, neurosyphilis -- according to these doctors.
I had multiple brain MRIs that revealed lesions in my brain; these were due to my high blood pressure, the doctors told me. I continued to get little headaches that would come, last for a minute and disappear. My legs would lose feeling and everyday it seemed like I was getting weaker. I would get Bell's palsy -- a syndrome where part of my face would be paralyzed and slump to one side. My body would twitch uncontrollably, and then tighten up to the point where I thought my bones would break.
I was told my pain was all in my head, that it wasn't real. Why you should be afraid of Lyme disease. I should have been thinking about Lyme disease. I know several friends and family members who have contracted the disease. But I had moved to South. I didn't think I had been bitten by a tick. I had no rash. During an appointment with my neurologist last month, after another trip to the emergency room, I asked: What about Lyme disease? I was devastated.
Had I only thought of it earlier, or had my doctors only thought of it earlier, the bacteria that manifests itself as the disease could have been eradicated. In the medical community, chronic Lyme disease is a controversial diagnosis. Lyme disease is usually treated with medication for no longer than 28 days.
But for some, even when the bacteria that causes the disease is gone, the symptoms still remain. The CDC goes on to say that no studies have proven antibiotic treatment past three to four weeks leads to any improvement in the patient. Some doctors disagree. Since there is still so much we don't know about the disease, the controversy ensues. In the next few months, I will see what relief my treatment will bring. My only hope is that by sharing my story, others won't suffer the same fate as me.